Coxsackievirus B4 and type 1 diabetes pathogenesis: contribution of animal models

Coxsackievirus B4 and type 1 diabetes pathogenesis: contribution of animal models

Source :

• Diabetes/Metabolism Research and Reviews [ 1520-7552 ] ; 2009-10.

English descriptors

• Teeft :
  • Allele, Amino acids, Animal models, Autoantibody, Autoantigen, Autoimmune, Autoimmune diabetes, Autoimmune disease, Autoimmune process, Autoimmune reaction, Autoimmunity, Autoreactive, Bbdp, Bbdp rats, Beta, Beta cells, Better knowledge, Biologie pathologie, Bystander, Bystander activation, Capsid protein, Capsid proteins, Cell destruction, Cell endocrinol, Cells destruction, Certain conditions, Chatterjee, Childhood diabetes, Childhood type, Chru lille, Clin, Clin pract, Copyright, Coxsackie, Coxsackie virus, Coxsackievirus, Coxsackievirus infection, Diabetes, Diabetes acceleration, Diabetes care, Diabetes development, Diabetes incidence, Diabetes installation, Diabetes mellitus, Diabetes metab, Diabetes onset, Diabetes pathogenesis, Diabetic, Diabetic animals, Diabetic ketoacidosis, Diabetic mice, Diabetic models, Diabetic patients, Diabetogenic, Diabetogenic phenotype, Diabetogenic process, Diabetogenic strain, Diabetologia, Direct cytolytic infection, Dual role, Dystrophia myotonica kinase, Enterovirus, Enterovirus infection, Enterovirus infections, Environmental factors, Exocrine pancreas, General population, Genetic background, Genetic factors, Genetic predisposition, Glutamate decarboxylase, Horwitz, Human beings, Human genes, Human pathology, Human species, Humoral response, Immune, Immune response, Immunization, Immunol, Immunological function, Important role, Infection, Infectious virus, Insulin, Insulitis, Intraperitoneal route, Islet, Islet antigens, Islet autoantigen, Islet autoantigens, Islet cells, Islet neogenesis, John wiley sons, Juvenile diabetes research foundation, Komeda, Lille, Locus, Lymphocyte, Lymphoid organs, Lymphoid tissue, Main animal models, Main results, Mellitus, Metab, Mice inoculated, Microbiol immunol, Mimicry, Molecular mimicry, Mouse, Mouse model, Mouse pancreas, Murine, Murine models, Murine pancreas, Murine splenic, Mutation, Oral route, Other hand, Other models, Other molecules, Pancreas, Pancreatic, Pancreatic cells, Pancreatic islet cells, Pancreatic islets, Pancreatic organ donors, Pancreatic tissue, Pathogenesis, Pathogenic mechanisms, Pathogenic process, Pathogenic processes, Peptide, Persistent infection, Persistent infections, Primary cultures, Proc natl acad, Protective role, Replication, Risk factor, Same model, Sarvetnick, Second infection, Spontaneous development, Spontaneous diabetes, Spontaneous diabetes mellitus, Study group, Successive passages, Susceptibility, Susceptible mice, Swiss mice, Thymic cells, Trace elements, Transgenic expression, Viral, Viral antigen, Viral genome, Viral infection, Viral infections, Viral pathogenesis, Viral persistence, Viral replication, Virol, Virus, Virus dissemination, Virus infection, Virus infections, Virus inoculation.

Abstract

• https://api.istex.fr/ark:/67375/WNG-C365ZBDB-L/fulltext.pdf

Affiliations:

• France, Tunisie
• Hauts-de-France, Nord-Pas-de-Calais
• Lille
• Université Lille 2

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